![]() ![]() The repression by ethylene is mediated by a direct regulation of the ethylene response transcription factor ETHYLENE INSENSITIVE3 (EIN3) on multiple SA biosynthesis and signaling genes. ![]() The inhibition of SA biosynthesis by ethylene, while mainly through ISOCHORISMATE SYNTHASE1/SALICYLIC ACID-INDUCTION DEFICIENT2 ( ICS1/ SID2) at normal temperature, is through ENHANCED DISEASE SUSCEPTIBILITY5 ( EDS5)/ SID1, ICS2, and ICS1/SID2 at lower temperature. SA signaling is repressed by jasmonic acid and ethylene at both normal and low temperatures. This enhancement is dependent on SA signaling and is accompanied by up-regulation of multiple SA biosynthesis and signaling genes at lower temperature. Here, we report that moderately low temperature enhances immunity to the bacterial pathogen Pseudomonas syringae in Arabidopsis ( Arabidopsis thaliana). Earlier studies identified intracellular immune receptor nucleotide-binding leucine-rich repeat (NLR) genes and salicylic acid (SA) as targets of high-temperature inhibition of plant immunity. ![]() Temperature has a large impact on plant immune responses. ![]()
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